LILIUM SP POISONING IN A FELINE

Autores

  • Júlia Maso Rissardi
  • Laura Dias da Silva
  • Lisiane Siqueira
  • Amanda Carolina Cole Varela
  • Cecília Mazutti Andrade
  • Emilly Baldissarelli
  • Gabriéla Witkowski Rutikoski
  • João Antônio Duarte Lampugnani
  • Héllen Alana de Castro
  • Pedro Henrique Simioni Dalla Rosa
  • Gabriele de Almeida
  • Daiane Rocha Rosa
  • Artur Primieri Pazinatto
  • Leonardo José Gil Barcellos

Palavras-chave:

Toxic plants, Acute renal failure, Nephrotoxicity

Resumo

INTRODUCTION: Lilies are ornamental plants that are part of the decoration of indoorand outdoor spaces. However, many people do not know that these plants have thepotential to be lethally toxic to cats, who, out of curiosity, accidentally ingest parts of theplant when cleaning themselves or playing with the flowers. Ingestion can cause acutekidney failure.CASE REPORT: A 3-month-old male feline weighing 1.6 kg and of mixed breed wastreated at the Veterinary Hospital of the University of Passo Fundo due to vomiting andanorexia. During the anamnesis, the owner reported that the animal had ingested lily theprevious day. Complete blood count, serum biochemistry (alkaline phosphatase,creatinine, phosphorus, urea, alanine aminotransferase), urinalysis, and ultrasound wereperformed. The complete blood count showed leukocytosis with neutrophilia and mildeosinophilia, without other alterations. Abdominal ultrasound showed hepatomegaly anddecreased echogenicity, suggesting hepatotoxicity. A marked medullary sign wasobserved in the left kidney, suggesting nephrotoxicity or tubular necrosis associated withintoxication, in addition to ureterolithiasis and clusters of sandstone stones. In the rightkidney, small areas of mineralization were observed in the pelvic recesses and amoderately dilated pelvis, as well as the presence of ureterolithiasis. The urinalysisshowed a clear, yellowish appearance and a urine specific gravity of 1.030. The chemicalexamination revealed the presence of three protein crosses and two occult blood crosses.The animal was treated with acetylcysteine 70 mg/Kg TID, ondansetron 1mg/Kg TID,omeprazole 1 mg/Kg SID, and prazosin hydrochloride 0.07 mg/Kg BID, all orally. Withthe evolution of the clinical condition, the feline was discharged.DISCUSSION: Treatment consists of assessing clinical signs, inducing emesis, andproviding supportive therapy, such as intravenous fluid therapy, gastric protectors andnutritional support. Diagnosis is based on clinical history, laboratory tests, and ultrasoundto confirm the clinical picture. It is the veterinarian's responsibility to advise owners aboutthe risks of certain plants to the health of their animals and to recommend the removal ofthese species from the environment to prevent potential poisoning.

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2025-10-03

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